Dr. Pramod Bhasme | Microbiology | Best Review Paper Award
McGowan Institute for Regenerative Medicine | India
Dr. Pramod Bhasme is a distinguished researcher specializing in microbiology, molecular biology, and regenerative medicine, with expertise in bacterial pathogenesis, biofilm regulation, and wound healing. He earned his B.Sc., M.Sc., and M.Phil. in Microbiology from Karnatak University Dharwad, India, where he studied bacteriocins and mastitis-related pathogens, before completing a Ph.D. in Microbiology at the University of Chinese Academy of Sciences, Beijing, under Prof. Ma Luyan. His doctoral research focused on quorum sensing, virulence regulation, and biofilm disruption in Pseudomonas aeruginosa. He later held a postdoctoral fellowship at Indiana University and currently serves as a Postdoctoral Associate at the McGowan Institute for Regenerative Medicine, University of Pittsburgh. His work explores electroceutical technologies, bacteriophage therapy, senescence biology, and wound-healing strategies using transgenic models. Dr. Bhasme’s research interests include quorum sensing inhibition, cyclic-di-GMP signaling, protein engineering, and macrophage–keratinocyte interactions, with a strong focus on translational medicine. A recipient of the CAS-TWAS President’s Fellowship, he has published in leading journals such as Nature Communications, ACS Nano, Environmental Microbiology, JoVE, and mLife. With 11 publications, 231 citations across 211 documents, and an h-index of 8, he is recognized as an emerging leader in infection biology and regenerative medicine.
Profile: Scopus | Google Scholar
Featured Publications
Sharma, A., Anthony, A. J., Gnyawali, S., Bhasme, P., et al. The mitochondrial bioenergetics of functional wound closure is dependent on macrophage to keratinocyte exosomal crosstalk. ACS Nano. (Accepted).
Zhang, Y., Bhasme, P., et al. Dual functions: A coumarin-chalcone conjugate inhibits cyclic-di-GMP and quorum sensing signaling. mLife, 2(3), 284–293.
El Masry, M., Bhasme, P., et al. Swine model of biofilm infection and invisible wound. Journal of Visualized Experiments, (196).
Pal, D., Ghatak, S., Bhasme, P., et al. Identification of a physiologic vasculogenic fibroblast state to achieve tissue repair. Nature Communications, 14(1), 1129.
Xu, A., Wang, D., Bhasme, P., et al. Mutations in WspA lock the Wsp signal system into an active state. Environmental Microbiology.